A recent study builds on prior work, citing Aagaard et al. (2026) in Molecular Psychiatry and earlier studies by Horner et al. (2025) and Borge et al. (2021, supporting diet-related associations and methodological considerations.

The article emphasizes considering heritability and non-transmitted genetic effects when researching ADHD, noting the need to control for genetic confounding.

Key references include Aagaard et al. (2026) on a genetic investigation of maternal diet and offspring ADHD, with supporting studies by Borge et al. (2021) and Horner et al. (2025) assessing diet-related associations.

The findings indicate that an offspring’s ADHD risk cannot be attributed to the mother’s prenatal diet alone, underscoring a strong genetic influence.

ADHD risk results from a combination of genetic predisposition and early environmental exposures, with genetics accounting for a large portion of heritability.

The new study design by Aagaard et al. (2026) uses a genetic model to separate genetic effects from environmental exposure, analyzing 700 mother-child pairs from mid-pregnancy to age 10.